Genetic engineering to enhance mercury phytoremediation

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Genetic engineering to enhance mercury phytoremediation.

Most phytoremediation studies utilize merA or merB genes to modify plants via the nuclear or chloroplast genome, expressing organomercurial lyase and/or mercuric ion reductase in the cytoplasm, endoplasmic reticulum or within plastids. Several plant species including Arabidopsis, tobacco, poplar, rice, Eastern cottonwood, peanut, salt marsh grass and Chlorella have been transformed with these g...

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Phytoremediation of organomercurial compounds via chloroplast genetic engineering.

Mercury (Hg), especially in organic form, is a highly toxic pollutant affecting plants, animals, and man. In plants, the primary target of Hg damage is the chloroplast; Hg inhibits electron transport and photosynthesis. In the present study, chloroplast genetic engineering is used for the first time to our knowledge to enhance the capacity of plants for phytoremediation. This was achieved by in...

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Genetic engineering strategies for enhancing phytoremediation of heavy metals

The industrial revolution has increased the use of metals for various processes and operations. The waste containing heavy metals are transported to the environment; air, water and soil through the various sources which has increased the burden in the environment. Phytoremediation has been found a promising, cost-effective, aesthetically pleasing, in situ treatment technology for the remediatio...

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Phytoremediation of mercury-contaminated soils by Jatropha curcas.

Jatropha curcas plants species were tested to evaluate their phytoremediation capacity in soils contaminated by different levels of mercury. The experimental treatments consisted of four levels of mercury concentrations in the soil - T0, T1, T5, and T10 (0, 1, 5, and 10 μg Hg per g soil, respectively). The total mercury content absorbed by the different plant tissues (roots, stems and leaves) w...

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ژورنال

عنوان ژورنال: Current Opinion in Biotechnology

سال: 2009

ISSN: 0958-1669

DOI: 10.1016/j.copbio.2009.02.010